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Your manuscript at www.biomedcentral.com/submitdoi:10.1186/s12866-014-0244-0 Rosiglitazone Cite this article as: Faria-Oliveira et al.: Methodologies to generate, extract, purify and fractionate yeast ECM for analytical use in proteomics and glycomics. BMC Microbiology 2014 14:244.
Song et al. Molecular Cancer 2014, 13:235 http://www.molecular-cancer.com/content/13/1/CORRECTIONOpen AccessCorrection: Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in gliomaYe Song1,2, Qisheng Luo1,3, Hao Long1, Zheng Hu1, Tianshi Que1, Xi'an Zhang1, Zhiyong Li1, Gang Wang1, Liu Yi1, Zhen Liu2,4*, WeiYi Fang2* and Songtao Qi1*Correction After the publication of this work [1] it was brought to the authors' attention that the U251-pLVTHM panel in Figure fiveB and the U251 negative control (NC) panel in Figure fiveD contained a duplication in error. The correct version of Figure five (Figure 1 here) is given below. The authors regret any inconvenience that this inaccuracy may have caused.Author details 1 Department of Neurosurgery, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, PR China. 2Cancer Research Institute of Southern Medical University, Guangzhou, Guangdong, PR China. 3 Department of Neurosurgery, Affiliated Hospital, Youjiang Medical College for Nationalities, Baise, Guangxi, PR China. 4Department of Pathology, Basic School of Guangzhou Medical College, Guangzhou, Guangdong, PR China. Received: 16 October 2014 Accepted: 16 October 2014 Published: 20 January 2015 Reference 1. Song Y, Luo Q, Long H, Hu Z, Que T, Zhang X, et al. Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma. Mol Cancer 2014, 13:65.doi:10.1186/1476-4598-13-235 Cite this article as: PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/10956013 Song et al.: Correction: Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma. Molecular Cancer 2014 13:235.Submit your next manuscript to BioMed Central and take full advantage of:?Convenient online submission ?Thorough peer review* Correspondence: narcissus_jane@163.com; fangweiyi1975@163.com; qisongtaosjwk@163.com Equal contributors 4 Department of Pathology, Basic School of Guangzhou Medical College, Guangzhou, Guangdong, PR China 2 Cancer Research Institute of Southern Medical University, Guangzhou, Guangdong, PR China 1 Department of Neurosurgery, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, PR China?No space constraints or color figure charges ?Immediate publication on acceptance ?Inclusion in PubMed, CAS, Scopus and Google Scholar ?Research which is freely available for redistributionSubmit your manuscript at www.biomedcentral.com/submit?2014 Song et al.; licensee BioMed Central. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.Song et al. Molecular Cancer 2014, 13:235 http://www.molecular-cancer.com/content/13/1/Page 2 ofFigure 1 Stably inhibited ENO1 expression decreases cell migration and invasion. (A). Stablydownregulating ENO reduced the migration ability of shENO1-U251 and shENO1-U87 cells in.

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